Dry Needling Article SRF Journal
Dry Needling Article SRF Journal
The perpetuating factors of myofascial pain syn- twitch of the muscle is the desired response; how-
dromes are low level musculature contraction, unac- ever, benefits can occur even without a twitch of the
customed eccentric contraction, muscle overloads, and muscle. The simple insertion of the needle into the
muscle fatigue. These factors can be caused by, but not taut bands can interrupt the pathogenic process and
limited to, mechanical dysfunctions such as forward produce mechanical changes in the tissue. (Figure 3)
head postures, joint hypermobilities, ergonomic stres- The insertion of a needle into the trigger point can
sors, poor body mechanics, and scoliosis. produce a deep ache or cramping pressure that lasts
Dry needling techniques are developed on various only briefly. Reproduction of pain and referral of
models, which are implemented in physical therapy symptoms may also occur, and soreness may last up
practices on a daily basis. Dr. Chan Gunn contributed to 24–48 hours. Improvements in functional range of
to the development of dry needling and introduced the motion, decrease in complaints of pain, and ease of
term intramuscular stimulation (IMS), in which he de- mobility may be seen after treatment.
scribed that myofascial pain syndromes are a result of Dry needling allows access to deep musculature
radiculopathy or peripheral neuropathy, causing a dis- that may never have been reached without the use
ordered function of the peripheral nerve. This concept of a needle. By releasing the myofascial restrictions
is referred to as the radiculopathy model and is based through trigger point dry needling, a physical therapist
on the Cannon and Rosenblueth’s law of denervation, is able to further enhance a patients care and return
which states that free flow of nerve impulses maintains to function. Trigger point dry needling can disrupt the
innervated function and integrity of structures. When
any neural flow is disrupted, all the structures that
are innervated by that nerve, such as skeletal muscle,
smooth muscle, spinal neurons, sympathetic ganglia,
and sweat glands, are affected and can become atro-
phic, highly irritable, and hypersensitive. The trigger
point model describes myofascial trigger points con-
sisting of taut musculature bands due to the excessive
release of acetylcholine. Myofascial trigger points are
classified as active or latent. Active myofascial trig-
ger points can cause local and referred pain, or other
parasthesias, whereas latent myofascial trigger points
may not produce pain without being stimulated. Ac-
tive myofascial trigger points typically refer pain to
a particular site, and these sites are not restricted to
a single segmental or peripheral nerve distribution.
Clinically, myofascial trigger points can cause motor
dysfunction or muscle weakness as a result of motor
inhibition, restricted motion, and muscular stiffness.
Furthermore, sensory dysfunctions may be noted
through localized tenderness, referral of pain to spe-
cific areas, hyperalgesia (extreme pain reaction to a
painful stimulus), and/or allodynia (pain reaction to a
non-painful stimulus).
The goal of the insertion of a fine filament needle
Figure 3. Identification of trigger by low-twitch response to palpa-
into a trigger point is to produce a twitch response tion. Image courtesy of Medscape.com, 2011, available at http://
(short contraction) of the muscle being needled. The emedicine.com/article/89095-overview.
39 Journal of The Spinal Research Foundation SPRING 2013 VOL. 8 No. 1
SPRING 2013