Hyperkalemia PDF
Hyperkalemia PDF
• Closely monitor the response to intravenous calcium glu- sodium bicarbonate solution can cause a shift of water into
conate, particularly in clients taking digitalis. Calcium in- the extracellular space.
creases the risk of digitalis toxicity. • Monitor clients receiving cation exchange resins and sorbitol
Risk for Imbalanced Fluid Volume for fluid volume excess. The resin exchanges potassium for
Renal failure is a major cause of hyperkalemia. Clients with sodium or calcium in the bowel. Excessive sodium and water
renal failure also are at risk for fluid retention and other elec- retention may occur.
trolyte imbalances. Using NANDA, NIC, and NOC
• Closely monitor serum potassium, BUN, and serum creatinine. Chart 5–4 shows links between NANDA nursing diagnoses, NIC,
Notify the physician if serum potassium level is greater than 5 and NOC when caring for a client with a potassium imbalance.
mEq/L, or if serum creatinine and BUN levels are increasing.
Serum creatinine and BUN are the primary indicators of renal Home Care
function. Levels of these substances rise rapidly in acute renal Preventing future episodes of hyperkalemia is the focus when
failure, more slowly in chronic renal failure (see Chapter 00). preparing the client for home care. Include the family, a signif-
• Maintain accurate intake and output records. Report an im- icant other, or a caregiver when teaching the following topics.
balance of 24-hour totals and/or urine output less than 30
mL/hour. Oliguria (scant urine) or anuria (no urine output) • Recommended diet and any restrictions including salt sub-
may indicate renal failure and an increased risk for hyper- stitutes and foods high in potassium
kalemia and fluid volume excess. • Medications to be avoided, including over-the-counter and
• Monitor clients receiving sodium bicarbonate for fluid vol- fitness supplements
ume excess. Increased sodium from injection of a hypertonic • Follow-up appointments for lab work and evaluation