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Table of Contents
Cover image
Title Page
Copyright
Further Reading
Preface
ECG 1
ECG 2
ECG 3
ECG 4
ECG 5
ECG 6
ECG 7
ECG 8
ECG 9
ECG 10
ECG 11
ECG 12
ECG 13
ECG 14
ECG 15
ECG 16
ECG 17
ECG 18
ECG 19
ECG 20
ECG 21
ECG 22
ECG 23
ECG 24
ECG 25
ECG 26
ECG 27
ECG 28
ECG 29
ECG 30
ECG 31
ECG 32
ECG 33
ECG 34
ECG 35
ECG 36
ECG 37
ECG 38
ECG 39
ECG 40
ECG 41
ECG 42
ECG 43
ECG 44
ECG 45
ECG 46
ECG 47
ECG 48
ECG 49
ECG 50
ECG 51
ECG 52
ECG 53
ECG 54
ECG 55
ECG 56
ECG 57
ECG 58
ECG 59
ECG 60
ECG 61
ECG 62
ECG 63
ECG 64
ECG 65
ECG 66
ECG 67
ECG 68
ECG 69
ECG 70
ECG 71
ECG 72
ECG 73
ECG 74
ECG 75
Part 2 More challenging ECGs
ECG 76
ECG 77
ECG 78
ECG 79
ECG 80
ECG 81
ECG 82
ECG 83
ECG 84
ECG 85
ECG 86
ECG 87
ECG 88
ECG 89
ECG 90
ECG 91
ECG 92
ECG 93
ECG 94
ECG 95
ECG 96
ECG 97
ECG 98
ECG 99
ECG 100
ECG 101
ECG 102
ECG 103
ECG 104
ECG 105
ECG 106
ECG 107
ECG 108
ECG 109
ECG 110
ECG 111
ECG 112
ECG 113
ECG 114
ECG 115
ECG 116
ECG 117
ECG 118
ECG 119
ECG 120
ECG 121
ECG 122
ECG 123
ECG 124
ECG 125
ECG 126
ECG 127
ECG 128
ECG 129
ECG 130
ECG 131
ECG 132
ECG 133
ECG 134
ECG 135
ECG 136
ECG 137
ECG 138
ECG 139
ECG 140
ECG 141
ECG 142
ECG 143
ECG 144
ECG 145
ECG 146
ECG 147
ECG 148
ECG 149
ECG 150
Index
Copyright
Notices
Practitioners and researchers must always rely on their own
experience and knowledge in evaluating and using any information,
methods, compounds or experiments described herein. Because of
rapid advances in the medical sciences, in particular, independent
verification of diagnoses and drug dosages should be made. To the
fullest extent of the law, no responsibility is assumed by Elsevier,
authors, editors or contributors for any injury and/or damage to
persons or property as a matter of products liability, negligence or
otherwise, or from any use or operation of any methods, products,
instructions, or ideas contained in the material herein.
ISBN 978-0-7020-7458-5
978-0-7020-7459-2
Printed in China
Last digit is the print number: 9 8 7 6 5 4 3 2 1
Further Reading
These symbols indicate cross-references to useful information in
The ECG Made Easy, 9th edition (Elsevier, 2019) and The ECG Made
Practical, 7th edition (Elsevier, 2019).
Preface
Learning about ECG interpretation from books such as The ECG Made
Easy or The ECG Made Practical (the previous editions of which were
called The ECG in Practice) is fine so far as it goes, but it never goes far
enough. As with most of medicine there is no substitute for
experience, and to make the best use of the ECG there is no substitute
for reviewing large numbers of them. ECGs need to be interpreted in
the context of the patient from whom they were recorded. You need to
learn to appreciate the variations of normality and of the patterns
associated with different diseases, and to think about how the ECG
can help patient management.
150 ECG Cases is the fifth edition of 150 ECG Problems. We have
changed the title to emphasize the importance of relating an ECG to
the patient from whom it was recorded: these 150 records came from
real patients, and at the time of recording were essential for the
diagnosis and management of real patients. The aim of the book is the
same as in the previous editions: it allows the inclusion of a lot more
ECGs than is possible in The ECG Made Easy and The ECG Made
Practical, and it is designed for the reader to improve his or her
understanding of ECGs by testing recognition skills. Some 10% of the
ECGs are new compared with the previous edition.
We have divided the book into two parts. The first part includes 75
ECGs that are commonly seen, and we have called this section
‘Everyday ECGs’ because these are the ECG patterns that crop up
frequently in the Accident & Emergency department or the outpatient
clinic. We have included several examples of ECGs from patients with
common problems, such as myocardial infarction. Those who have
read The ECG Made Easy should be able to recognize most of these
ECGs. The second part of the book, which we have called ‘More
challenging ECGs’ includes records seen less often, some of which
could be described as ‘difficult’ – and in some the reader may disagree
with our interpretation. But on the whole, anyone who has read The
ECG Made Practical should get most of the ECGs in Part 2 right.
JH, DA, JH
Introduction: making the most of
the ECG
Recording and reporting an ECG should never be an end in itself. The
ECG is a basic and valuable tool in the investigation of cardiac
problems, and it can be helpful in the case of non-cardiac problems,
too, but it must always be viewed in the context of the patient from
whom the record came. The ECG must never be a substitute for taking
a proper medical history and carrying out a careful physical
examination. Because it is simple, harmless and cheap, the ECG is
usually the first investigation in a patient with possible cardiac
disease, and it may be followed by the plain chest X-ray, the
echocardiogram, radionuclide studies, computed tomography (CT),
magnetic resonance imaging (MRI), and cardiac catheterization and
angiography – but none of these are substitutes. The ECG, a recording
of the electrical activity of the heart, gives information that can be
obtained in no other way. However, even though it is irreplaceable, it
is not infallible.
ECGs are recorded from a wide variety of patients, in an attempt to
help with a wide variety of possible diagnoses. An ECG is frequently
recorded in the course of ‘health screening’, but here it must be
regarded with considerable caution. It cannot be assumed that
individuals who present themselves for screening are asymptomatic –
the process may be being used as a substitute for a consultation with a
doctor. The ECG itself may cause difficulties of interpretation, for
there are a dozen or more normal variants. Minor abnormalities, such
as non-specific ST segment or T wave changes, will have diagnostic
and prognostic significance if the individual has symptoms that may
be cardiac in origin, but these changes can be of no importance in
totally healthy people. It is rare for an ECG to demonstrate anything
of importance in a totally healthy individual, although in athletes the
detection of abnormalities suggesting asymptomatic hypertrophic
cardiomyopathy is important.
In patients with chest pain, the ECG is important but sometimes
misleading. It is essential to remember that the ECG can remain
normal for some hours after the onset of a myocardial infarction. Too
often patients are sent home from an A&E department because their
ECG is normal, despite a reasonably convincing story of ischaemic
chest pain. Under such circumstances the ECG should be repeated
several times to see if changes are appearing, and patient management
should depend on the plasma troponin level rather than on the ECG.
Nevertheless, the ECG is important for deciding treatment in a patient
with chest pain, for the management of a patient with myocardial
infarction with ST segment elevation is quite different from that of a
patient whose ECG shows a non-ST segment elevation infarction.
Patients with intermittent chest pain that could be angina frequently
have completely normal ECGs at rest – and then the exercise test can
be valuable. The exercise test is to some extent being replaced by
myocardial perfusion scanning for the diagnosis of coronary disease
because its predictive accuracy depends on the likelihood of the
patient having angina, because there can be false negative or false
positive results, and because exercise tests are sometimes unreliable in
women. Remember that an exercise test is safe, but not totally safe,
because arrhythmias (including ventricular fibrillation) may be
induced. Nevertheless, the exercise test has the great advantage of
showing a patient's exercise tolerance, and also showing what limits
his capability.
The ECG also has a role in the investigation of patients with
breathlessness, for it can show changes associated with heart disease
(e.g. an old myocardial infarction) or with chronic chest disease.
Evidence of left ventricular hypertrophy may point to hypertension,
mitral regurgitation or aortic stenosis or regurgitation, and right
ventricular hypertrophy may be the result of pulmonary emboli or
mitral stenosis – however, all of these should have been detected
during the examination of the patient. The ECG is not a good tool for
grading the hypertrophy of the different heart chambers. It is
particularly important to remember that the ECG cannot demonstrate
heart failure: it may suggest a condition that may cause heart failure,
but it is impossible to determine from an ECG whether or not a
patient is in heart failure. However, in the presence of a completely
normal ECG, heart failure is certainly unlikely.
There are characteristic ECG appearances in several conditions that
are not primarily cardiac – for example with severe electrolyte
derangement. ECG monitoring is not an acceptable way of following
electrolyte changes in conditions such as diabetic ketoacidosis, but at
least any abnormalities may prompt the appropriate biochemical tests.
The ECG has, however, become important in the development of new
drugs, for any drug that causes QT prolongation – and this is by no
means uncommon – may cause sudden death due to ventricular
tachycardia.
It is in the investigation and management of patients with possible
arrhythmias that the ECG is of paramount importance. Patients may
complain of palpitations or dizziness and syncope as a result of
rhythm disturbances, and there is no way of identifying these with
certainty other than with an ECG. Dizziness and syncope can be the
result of rhythms that are either too fast or too slow for an effective
cardiac output, or of slow rhythms associated with disorders of
conduction. There may be little in the patient's history to point
specifically to a cardiac problem when dizziness or collapse is the
main symptom, but an appropriately abnormal ECG may
immediately point to the right diagnosis. When a patient complains of
palpitations there is a clearly a heart problem of some sort, and it is
usually possible to come close to a diagnosis by taking a careful
history – the patient with extrasystoles will describe the heart
‘jumping out of the chest’ or something equally unlikely, and the
problem will be worse when lying down at night, and after smoking
and alcohol. The patient with a true paroxysmal tachycardia will
describe the sudden onset (and sometimes the sudden cessation) of
the rapid heartbeat, and if the attack is associated with chest pain,