Types of Ecg 1. Resting ECG (12-Lead ECG)
Types of Ecg 1. Resting ECG (12-Lead ECG)
3. Event Monitor
• Purpose: Similar to a Holter monitor, but used for longer durations, typically 30 days
or more.
• Description: An event monitor is worn by the patient for a longer period (typically up
to 30 days). It records only when activated by the patient when they experience symptoms
(like palpitations or dizziness).
• Indications: Intermittent symptoms that are hard to correlate with a short ECG or
Holter recording, such as occasional arrhythmias.
4. Stress ECG (Exercise ECG or Treadmill Test)
• Purpose: Monitors both ECG and blood pressure over an extended period.
• Description: This combined test records the patient’s heart activity and blood
pressure, typically over 24 hours. This can help evaluate the relationship between blood
pressure changes and ECG changes, especially in patients with hypertension or those
experiencing symptoms like dizziness.
• Indications: Evaluation of arrhythmias and blood pressure fluctuations, especially in
hypertensive patients or those with unexplained symptoms.
8. Pre-Operative ECG
• Purpose: Used for monitoring symptoms and identifying abnormal heart rhythms over
extended periods.
• Description: This device is similar to a Holter monitor but often used for longer
periods, especially for individuals with infrequent symptoms. The patient can activate the
device when they feel symptoms (such as palpitations), and it will record the ECG during that
event.
• Indications: Intermittent symptoms such as palpitations, fainting, or dizziness that do
not occur frequently enough to be caught by a routine ECG.
• Confirm patient identity using two identifiers (e.g., name and date of birth) to ensure
accuracy and compliance with safety protocols.
• Verify the ECG order and confirm the type of ECG requested (e.g., 12-lead, 3-lead) to
prepare for the correct setup.
• Explain the procedure to the patient, including its purpose, what to expect, and how it
may feel (e.g., mild discomfort when removing electrodes).
• Address any questions or concerns the patient may have to reduce anxiety, which
could cause restlessness and impact ECG accuracy.
• Assess the patient for skin conditions or sensitivities (e.g., allergies to adhesives or
skin irritation) and prepare alternative options if needed.
• Identify any recent chest surgeries, wounds, or pacemaker implants, which may
require adjusting electrode placement.
• Ensure the patient can comfortably lie in a supine position and remain still during the
ECG, as movement can cause artifacts on the reading.
• Make adjustments if the patient has difficulty lying flat (e.g., semi-Fowler’s position if
there are respiratory concerns) but inform the provider as position changes may impact
results.
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5. Privacy and Environment
• Provide privacy by closing curtains or doors and covering any parts of the body not
involved in the procedure.
• Ensure the room is free from electrical interferences (e.g., mobile devices or other
electronic devices) as they can cause ECG artifacts.
6. Skin Preparation
• Clean the skin with alcohol wipes to remove oils or lotions that can reduce electrode
adherence.
• If the patient has significant chest hair, clip (do not shave) the area to improve
electrode contact without causing skin irritation.
7. Equipment Readiness
• Confirm that the ECG machine is functioning correctly, has sufficient paper, and is set
to the appropriate settings for the test.
• Prepare electrodes, leads, and any additional supplies, such as conductive gel if non-
adhesive electrodes are used.
Equipment Needed
Assessment Steps
Implementation Steps
• Determine the reason for the ECG (e.g., baseline or diagnostic for chest pain).
• Assess the patient’s understanding of the procedure and ability to follow instructions.
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• Perform hand hygiene, ensure privacy, and position the patient in a supine position.
• Clean the skin with alcohol wipes at the electrode placement sites, and, if necessary, clip any chest hair (do not
shave).
3. Placing Electrodes
• Chest leads:
• Limb leads:
• Right wrist (aVr), Left wrist (aVl), Left ankle (aVf), Right ankle (ground).
• Turn on the ECG machine, enter patient demographic details, and ensure proper attachment of leads.
• Instruct the patient to lie still, avoid talking, and keep legs uncrossed.
• Press the button to record the ECG. If the tracing is unclear, verify connections and electrode contact.
5. Final Steps
• Detach the leads, remove electrodes carefully, and explain that the procedure is complete.
• Label the ECG with the patient’s details (name, age, gender, date, and time).
• Atrial Fibrillation: Irregular and often rapid heart rate, with absence of P waves and irregular QRS complexes.
• Ventricular Tachycardia: Fast, regular heart rate originating in the ventricles, with wide QRS complexes.
• Ventricular Fibrillation: Rapid, chaotic electrical activity, resulting in an uncoordinated contraction of the ventricles.
• ST-Segment Elevation: Indicates acute myocardial infarction (heart attack); the elevation in the ST segment usually
occurs in specific leads depending on the affected area of the heart.
• ST-Segment Depression: Often a sign of ischemia or angina, indicating reduced blood flow to the heart muscle.
3. Heart Block
• First-Degree AV Block: Prolonged PR interval (>200 ms), but all impulses are conducted from the atria to the
ventricles.
• Mobitz I (Wenckebach): Progressive lengthening of the PR interval until a QRS complex is dropped.
• Third-Degree AV Block (Complete Heart Block): No conduction between the atria and ventricles, resulting in
independent atrial and ventricular rhythms.
• Left Bundle Branch Block (LBBB): Wide QRS complex (>120 ms), with a notched or slurred R wave in leads V5 and
V6.
• Right Bundle Branch Block (RBBB): Wide QRS complex with an “RSR’” pattern (rabbit ears) in V1 and V2.
• Left Ventricular Hypertrophy (LVH): Tall R waves in the left chest leads (V5 and V6) and deep S waves in the right
chest leads (V1 and V2).
• Right Ventricular Hypertrophy (RVH): Large R wave in V1 and deep S wave in V6.
6. Electrolyte Imbalances
• Hyperkalemia: Tall, peaked T waves, flattened P waves, and widening of the QRS complex.
7. Pericarditis
• Global ST Elevation: Diffuse (widespread) ST elevation across multiple leads, often with PR depression.
ECG can provide signs that may indicate a patient is experiencing shock, though
it is not definitive on its own. Shock affects the entire cardiovascular system and
can result from various causes (e.g., hypovolemic, cardiogenic, septic, or
obstructive shock). On an ECG, here are some indicators that might be seen in a
patient experiencing shock, especially cardiogenic shock:
1. Tachycardia
• A fast heart rate (tachycardia) often appears as a compensatory response to
maintain cardiac output. Sinus tachycardia is commonly seen in shock, as the
heart tries to pump blood faster to meet the body’s needs.
2. Low Voltage QRS Complexes
• Reduced amplitude of the QRS complexes may indicate poor cardiac
output or pericardial effusion, especially in cardiogenic shock or cardiac
tamponade.
3. ST-Segment Changes
• ST-segment elevation or depression may suggest myocardial ischemia or
infarction, a possible cause of cardiogenic shock. Acute myocardial infarction
(AMI) can reduce the heart’s pumping ability, leading to shock if severe.
4. Arrhythmias
• Arrhythmias, such as ventricular tachycardia or ventricular fibrillation, can
indicate severe cardiac dysfunction, often seen in cardiogenic shock. These
arrhythmias may impair blood flow, worsening the shock state.
5. Signs of Myocardial Strain
• ECG changes like T-wave inversions or strain patterns may indicate
myocardial stress, particularly if the patient is in shock due to severe
hypertension, pulmonary embolism, or heart failure.
6. Electrical Alternans
• If cardiac tamponade is causing obstructive shock, you might see electrical
alternans (alternating QRS complex height) due to the heart’s movement within a
fluid-filled pericardial sac.
Limitations
While an ECG can provide clues, it is usually combined with clinical assessment
and other diagnostics (e.g., blood pressure, pulse, lactate levels,
echocardiogram) to confirm shock. Shock is primarily diagnosed by assessing vital
signs, signs of organ hypoperfusion (such as altered mental status or low urine
output), and blood tests indicating metabolic acidosis or elevated lactate levels.
Nursing interventions for patients experiencing different types of shock are aimed at
stabilizing the patient, supporting organ function, and addressing the underlying cause.
1. Cardiogenic Shock
• Assessments: Monitor ECG for arrhythmias, continuously assess vital signs, pulse
oximetry, urine output, and mental status to detect worsening perfusion.
• Oxygenation: Administer oxygen to maintain adequate oxygenation (e.g., SpO2 >
94%). If oxygenation remains inadequate, prepare for advanced airway management.
• IV Access and Medication: Ensure large-bore IV access for administering fluids and
medications.
• Vasopressors (e.g., norepinephrine, dopamine) may be given to improve blood
pressure and perfusion.
• Inotropes (e.g., dobutamine) can be used to improve myocardial contractility.
• Positioning: Place the patient in semi-Fowler’s position to reduce the workload on the
heart if blood pressure is stable. Avoid Trendelenburg, as it can increase cardiac workload.
• Limit Activity: Reduce physical exertion to decrease myocardial oxygen demand.
• Emotional Support: Offer reassurance to reduce anxiety, which can elevate heart
rate and oxygen demand.
• Notify Provider: Report any changes in cardiac status or failure to respond to
interventions.
2. Obstructive Shock
3. Hypovolemic Shock
• Assessments: Monitor vital signs closely (particularly blood pressure, heart rate),
assess for signs of worsening perfusion (e.g., cool skin, decreased urine output).
• Fluid Resuscitation: Initiate rapid IV fluid replacement with isotonic crystalloids (e.g.,
normal saline or lactated Ringer’s solution) to restore intravascular volume.
• Positioning: Place in a modified Trendelenburg position (legs elevated) to improve
venous return and support blood pressure.
• Monitor for Response to Fluids: Assess for improvement in blood pressure, heart
rate, urine output, and mental status.
• Control Bleeding: If the cause is blood loss, apply pressure to external bleeding sites
or prepare the patient for surgical intervention if needed.
• Oxygenation: Administer oxygen to maintain SpO2 levels, supporting cellular
oxygenation as perfusion is restored.
• Notify Provider: Alert the provider about any ongoing or worsening hypotension or
signs of inadequate perfusion.
• Septic Shock:
• Assessments: Closely monitor vital signs, mental status, and signs of organ
dysfunction.
• Antibiotics: Administer antibiotics promptly (within 1 hour of diagnosis) as ordered to
treat the infection.
• Fluid Resuscitation: Administer IV fluids to restore perfusion; monitor response
carefully.
• Vasopressors: If fluids are insufficient to maintain blood pressure, administer
vasopressors (e.g., norepinephrine).
• Monitor for Organ Dysfunction: Watch for worsening respiratory, renal, and
cardiovascular status, as sepsis can lead to multi-organ dysfunction.
• Anaphylactic Shock:
• Assessments: Monitor for airway obstruction, breathing difficulties, and signs of low
perfusion.
• Epinephrine: Administer IM epinephrine immediately to counteract severe allergic
reactions.
• Oxygenation: Provide high-flow oxygen, and prepare for advanced airway support if
necessary.
• Medications: Administer antihistamines (e.g., diphenhydramine) and corticosteroids
as ordered to reduce inflammatory response.
• IV Fluids: Provide fluids to counteract hypotension as needed.
• Frequent Monitoring: Reassess vital signs, oxygen saturation, ECG, and mental
status frequently.
• Emotional Support: Reassure the patient and family, and keep them informed.
• Documentation: Record all interventions, assessments, and changes in the patient’s
status meticulously to support continuous care.
Each type of shock requires timely, targeted interventions to stabilize the patient and prevent
organ damage.
The types of medications given to patients in shock depend on the underlying cause, as each
type of shock requires specific interventions. Intubation and bronchodilator use may also be
required depending on the shock type and the patient’s respiratory status. Here’s a
breakdown:
1. Cardiogenic Shock
• Medications:
• Inotropes (e.g., dobutamine) to increase the strength of cardiac contractions.
• Vasopressors (e.g., norepinephrine or dopamine) to maintain blood pressure if
needed.
• Diuretics (e.g., furosemide) may be used cautiously in cases with fluid overload,
especially if the patient has heart failure.
• Antiplatelets or anticoagulants (e.g., aspirin, heparin) if the shock is due to
myocardial infarction (MI).
• Antiarrhythmics (e.g., amiodarone) for arrhythmias contributing to shock.
• Intubation: Patients may be intubated if there is severe respiratory distress or
pulmonary edema, often seen in cardiogenic shock.
• Bronchodilators: Typically not used in cardiogenic shock, as respiratory issues are
usually secondary to cardiac dysfunction rather than bronchoconstriction.
2. Obstructive Shock
• Medications:
• Anticoagulants (e.g., heparin) or thrombolytics (e.g., alteplase) for pulmonary
embolism to dissolve or prevent further clotting.
• Vasopressors (e.g., norepinephrine) to maintain blood pressure and perfusion.
• Analgesics and sedatives if procedures are required (e.g., pericardiocentesis for
cardiac tamponade).
• Intubation: Often required if there is significant respiratory compromise, especially in
cases of pulmonary embolism.
• Bronchodilators: May be used if pulmonary embolism is causing bronchoconstriction
or if there is an underlying lung condition like asthma.
3. Hypovolemic Shock
• Medications:
• IV Fluids: Isotonic crystalloids (e.g., normal saline or lactated Ringer’s) are the
primary intervention for fluid resuscitation.
• Blood Products: Packed red blood cells, platelets, or plasma may be given in cases
of blood loss.
• Vasopressors: Used cautiously and only after fluid resuscitation, as they are less
effective when intravascular volume is low.
• Intubation: Usually not required unless hypovolemic shock has caused severe
hypoxia or respiratory distress.
• Bronchodilators: Not typically used, as bronchoconstriction is not an issue in
hypovolemic shock.
• Septic Shock:
• Antibiotics: Broad-spectrum antibiotics are administered as soon as possible.
• IV Fluids: Initial resuscitation with crystalloids to improve blood pressure and
perfusion.
• Vasopressors (e.g., norepinephrine) to maintain blood pressure when fluid
resuscitation is insufficient.
• Inotropes: May be added if there is myocardial dysfunction.
• Corticosteroids: Occasionally used if there is adrenal insufficiency or refractory
shock.
• Intubation: Patients may require intubation if they develop respiratory failure or acute
respiratory distress syndrome (ARDS) due to sepsis.
• Bronchodilators: Not typically used, unless there is underlying bronchoconstriction
due to pre-existing conditions.
• Anaphylactic Shock:
• Epinephrine: First-line treatment for anaphylaxis; given intramuscularly for immediate
effect.
• Antihistamines (e.g., diphenhydramine) to counteract histamine release.
• Corticosteroids (e.g., methylprednisolone) to reduce inflammation and prevent
delayed reactions.
• IV Fluids: For volume support if hypotension is present.
• Vasopressors (e.g., norepinephrine) if hypotension persists despite fluids and
epinephrine.
• Intubation: Often required if there is severe airway obstruction or respiratory distress.
• Bronchodilators: Used (e.g., albuterol) to relieve bronchospasm and improve airway
patency in anaphylaxis.
• Neurogenic Shock:
• Vasopressors (e.g., norepinephrine, dopamine) to restore vascular tone and maintain
blood pressure.
• IV Fluids: Administered carefully to maintain blood pressure.
• Intubation: May be required if there is significant respiratory depression due to spinal
cord injury or neurological compromise.
• Bronchodilators: Generally not used, as bronchoconstriction is not a typical feature
of neurogenic shock.
In the emergency room (ER), ECGs are crucial for the rapid assessment of patients with
suspected cardiac issues and other conditions that may affect the heart. Here’s a breakdown
of why ECGs are used in the ER, common emergencies that may prompt an ECG, and the
nursing interventions and medications associated with these emergencies.
1. Quick Diagnosis of Cardiac Events: ECGs provide immediate insights into heart
rhythm and electrical activity, helping diagnose conditions such as myocardial infarction (heart
attack), arrhythmias, and cardiac ischemia.
2. Monitoring for Life-Threatening Changes: Patients with chest pain, shortness of
breath, or palpitations can be monitored for signs of deterioration or impending cardiac
events.
3. Evaluation of Non-Cardiac Conditions: Certain non-cardiac emergencies, like
electrolyte imbalances or pulmonary embolism, can show distinct patterns on an ECG that
guide diagnosis and treatment.
1. Quick Diagnosis of Cardiac Events: ECGs provide immediate insights into heart
rhythm and electrical activity, helping diagnose conditions such as myocardial infarction (heart
attack), arrhythmias, and cardiac ischemia.
2. Monitoring for Life-Threatening Changes: Patients with chest pain, shortness of
breath, or palpitations can be monitored for signs of deterioration or impending cardiac
events.
3. Evaluation of Non-Cardiac Conditions: Certain non-cardiac emergencies, like
electrolyte imbalances or pulmonary embolism, can show distinct patterns on an ECG that
guide diagnosis and treatment.
Summary
The ECG in the ER provides essential, time-sensitive information for diagnosing and
managing cardiac and related emergencies. Nursing interventions focus on stabilizing the
patient, ensuring adequate oxygenation, and preventing complications, while medications are
chosen based on the specific emergency and patient condition.
.
1. Using the 6-Second Method (for Regular or Irregular Rhythms)
• This is the easiest and most commonly used method, especially for irregular rhythms.
• Step-by-Step:
1. Print or observe a 6-second ECG strip. On most ECG paper, each large box
represents 0.2 seconds, and 30 large boxes represent 6 seconds.
2. Count the number of R waves (the peaks in the QRS complex) in the 6-second strip.
3. Multiply that number by 10 to get the heart rate in beats per minute (bpm).
• Example: If you count 8 R waves in 6 seconds, the heart rate is 8 x 10 = 80 bpm.
2. Using the 300, 150, 100 Method (for Regular Rhythms Only)
• This method works well for regular rhythms and uses the large boxes on the ECG
paper.
• Step-by-Step:
1. Identify an R wave that aligns with a large box.
2. Count the number of large boxes between that R wave and the next R wave.
3. Use the following sequence to estimate the heart rate: 300, 150, 100, 75, 60, 50.
• 1 large box between R waves = 300 bpm
• 2 large boxes = 150 bpm
• 3 large boxes = 100 bpm
• 4 large boxes = 75 bpm
• 5 large boxes = 60 bpm
• 6 large boxes = 50 bpm
• Example: If there are 3 large boxes between R waves, the heart rate is approximately
100 bpm.
• Many cardiac monitors in hospitals automatically compute the heart rate by analyzing
the R-R interval.
• Steps:
1. Ensure that electrodes are placed correctly and securely on the patient.
2. Check that the monitor is set to calculate and display the heart rate (usually in bpm).
3. Observe the heart rate reading on the monitor, which is continuously updated.
Types of Normal Waves in ECG
1. P Wave
• Description: Represents atrial depolarization (the electrical impulse that causes the
atria to contract).
• Normal Characteristics: Smooth, rounded, and usually upright in most leads (except
in aVR).
• Duration: Less than 0.12 seconds (3 small squares).
2. QRS Complex
• Description: Represents ventricular depolarization (the electrical impulse causing the
ventricles to contract).
• Normal Characteristics: Sharp, narrow, and of short duration.
• Duration: 0.06 to 0.10 seconds (1.5 to 2.5 small squares).
• Components:
• Q Wave: Initial negative deflection before the R wave.
• R Wave: The first positive deflection following the P wave.
• S Wave: The negative deflection after the R wave.
3. T Wave
• Description: Represents ventricular repolarization (the recovery phase after
contraction).
• Normal Characteristics: Rounded and upright in most leads, typically in the same
direction as the QRS complex.
• Duration: Variable but usually about 0.16 seconds.
4. U Wave (may or may not be present)
• Description: Small wave that sometimes follows the T wave, often associated with the
final phase of ventricular repolarization.
• Normal Characteristics: Small, upright, and rounded.
• Clinical Relevance: Prominent U waves can sometimes be seen in cases of
hypokalemia or bradycardia.
PR Interval
• Description: Represents the time from the onset of atrial depolarization to the onset
of ventricular depolarization.
• Normal Duration: 0.12 to 0.20 seconds (3-5 small squares).
6. QT Interval
• Description: Represents the total time for ventricular depolarization and
repolarization.
• Normal Duration: Varies with heart rate, but typically 0.36 to 0.44 seconds.
7. ST Segment
• Description: The period between ventricular depolarization and repolarization.
• Normal Characteristics: Usually flat (isoelectric), indicating no active electrical change.
1. Abnormal P Waves
• Tall or Peaked P Waves: Seen in right atrial enlargement (P pulmonale).
• Broad, Notched P Waves: Seen in left atrial enlargement (P mitrale).
• Inverted P Waves: Indicate an ectopic atrial rhythm, such as atrial arrhythmia.
2. Abnormal QRS Complex
• Wide QRS Complex: Indicates delayed ventricular conduction, seen in bundle branch
blocks or ventricular rhythms.
• Pathologic Q Waves: Deep and wide Q waves can indicate myocardial infarction
(MI), representing dead or scarred myocardial tissue.
3. Abnormal T Waves
• Tall, Peaked T Waves: Often indicate hyperkalemia (elevated potassium levels).
• Flattened or Inverted T Waves: May indicate ischemia, hypokalemia, or pericarditis.
• Biphasic T Waves: Sometimes associated with ischemia or electrolyte imbalances.
4. ST Segment Abnormalities
• ST Elevation: Indicates myocardial injury, often seen in acute myocardial infarction
STEMI).
• ST Depression: Suggests myocardial ischemia, seen in cases of angina or non-ST-
elevation myocardial infarction (NSTEMI).
5. Abnormal U Waves
• Prominent U Waves: Can indicate hypokalemia, bradycardia, or conditions like left
ventricular hypertrophy.
• Inverted U Waves: May be seen in cases of ischemia or heart disease.
6. Abnormal PR Interval
• Short PR Interval: May suggest a pre-excitation syndrome like Wolff-Parkinson-White
(WPW) syndrome.
• Prolonged PR Interval: Seen in first-degree atrioventricular (AV) block, indicating
delayed conduction from the atria to the ventricles.
7. Abnormal QT Interval
• Prolonged QT Interval: May indicate risk for arrhythmias like Torsades de Pointes,
often caused by electrolyte imbalances, medications, or genetic conditions.
• Shortened QT Interval: Can be seen in hypercalcemia or certain genetic conditions.