How To Read Ecg Strips
How To Read Ecg Strips
Sympathetic nerve stimulation triggers the release of norepinephrine, At the same time, the impulse travels to the left atrium by
which increase the heart rate, increase SA node discharge, accelerates AV Bachmann’s bundle (interatrial of the tissue extending from the SA node to
node conduction time, and increase the force of myocardial contraction the left atrium). Transmission through the right and left atria occurs so
and cardiac output. Parasympathetic (vagal) stimulation triggers the rapidly that the atria contract almost simultaneously.
release of acetylcholine, which produces the opposite effects. The rate of
SA node discharge is decreased, thus slowing heart rate and conduction
through the AV node and reducing cardiac output.
INDICATIONS
The ECG is a useful tool in the diagnosis of those conditions that may cause
aberrations in the electrical activity of the heart. Examples of these
Transmission of heart’s impulse to a graphic display by ECG machine. The
conditions are as follows:
electrodes that are capable of conducting electrical activity from the heart
to the ECG machine are placed at strategic positions on the extremities and 1. MI and other types of coronary artery diseases, such as angina
chest precordium. 2. Cardiac dysrhythmias
3. Cardiac enlargement
3. The electrical energy sensed is then converted to a graphic by the 4. Electrolyte disturbances, especially of calcium and potassium levels
ECG machine. This display is referred to as the electrocardiogram 5. Inflammatory diseases of the heart
6. Effect on the heart by drugs such as digoxin (Lanoxin) and tricyclic
4. A heart contraction is represented by wave forms on the ECG graph
antidepressants
paper, which are designated P, Q, R, S and T waves
ECG LEADS AND NORMAL WAVE FORM INTERPRETATION produces very high R waves because the hypertrophied muscles
requires a stronger electrical current to depolarize
1. The standard ECG consists of 12 leads (I, II, III, AVR, AVL, AVF, V1,
V2, V3, V4, V5, V6)
a. Each lead records the heart’s electrical activity from a different
anatomic position
b. Identifies specific myocardial changes on certain leads; assist in
defining pathologic conditions
2. The normal amplitude of the P wave is 3mm or less; the P waves
that exceed these measurements are considered to be a deviation
from normal
3. The P-R interval is measured from the upstroke of the P wave to
the Q-R junction and is normally between 0.12 and 0.20 second
a. The P-R interval represents the time of impulse transmission from
the sinoatrial (SA) node to the atrioventricular (AV) node
b. There is a built-in delay in time at the AV node to allow for
adequate ventricular filling to maintain normal stroke volume (the
amount of blood ejected with each contraction)
5. The S-T segment begins at the end of the S wave, the first negative
4. The QRS complex contains separate waves and segments, which deflection after the R wave, and terminated at the upstroke of the T
should be evaluated separately. Normal QRS complex should be wave
between 0.06 and 0.10 second
6. The T wave represents the repolarization of myocardial fibers or
a. The Q wave, or first downward stroke after the P wave, is usually provides the resting state of myocardial work; the T wave should
less than 3 mm in depth. A Q wave of significant deflection is not always be present
normally present in the healthy heart. The pathologic Q wave
a. Normally, the T wave should not exceed a 5 mm amplitude in all
usually indicates a completed MI.
leads except the precordial (V1 to V6 leads, where it may be as high
b. The R wave is the first positive deflection after the P wave, normally as 10mm)
5-10 mm in height. Increases and decreases in amplitude become
significant in certain disease states. Ventricular hypertrophy
INTERPRET ECG. DEVELOP A SYSTEMATIC APPROACH TO ASSIST IN
ACCURATE INTERPRETATION FOR DYSRHYTHMIAS, MYOCARDIAL
DAMAGE, OTHER CHANGES
a. Determine the rate, is it fast, slow, normal?
i. A gross determination of rate can be accomplished by counting the
number of QRS complexes within a 6-second time interval (use the
superior margin of ECG paper) and multiplying the complexes by a
factor of 10. Note: One must be cautioned that this method is
accurate only rhythms that are occurring at normal intervals and
should not be used for determining rate always counted for 1 full
minute for accuracy
ii. Another means of obtaining rate is to divide the number of large
five-square blocks between each two QRS complexes into 300.
Three hundred large blocks represent 1 complexes #5 and #6
equals 5, or of 60
iii. (Sequence Method) For ventricular rate, find R wave that peaks on
a heavy black line and assign the following numbers to the next six
heavy black lines: 300, 150, 100, 75, 60, and 50. Then find the next
R wave peak and estimate the ventricular rate
SINUS BRADYCARDIA
Paroxysmal atrial tachycardia
PREMATURE ATRIAL CONTRACTION
MANAGEMENT
1. Generally requires no treatment
2. PACs should be monitored for increasing frequency
ANALYSIS (ATRIAL FLUTTER)
P wave: not present; instead, it is replaced by a saw-toothed pattern that
is produced by the rapid firing of the atrial focus. These waves are also
referred to as “F” waves
P-R interval: not measurable
QRS complex: Normal configuration and normal conduction time
T-wave: present but may be obscured by flutter waves
VTACH
MOBITZ 1