Patient Name : Mrs.RASHMI TRIPATHI Reg.

Date : 04/Oct/2024 08:42AM

Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:48AM
LAB No : PN160759 Reported : 04/Oct/2024 11:50AM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

DEPARTMENT OF HAEMATOLOGY
Investigation Name Result Unit Bio. Ref. Range Method

CBC With ESR(WHOLE BLOOD AND SODIUM CITRATE)

HAEMOGLOBIN, Whole Blood EDTA 11.1 g/dL 12-15 SLS-Hemoglobin
TOTAL LEUCOCYTE COUNT (TLC), Whole 6.38 thousand/cmm 4.0 - 10.0 Flow Cytometry
Blood EDTA
Differential Leucocyte Count (DLC)
NEUTROPHILS 43.10 % 40.0 - 80.0 Flow Cytometry /
Microscopy
LYMPHOCYTES 43.30 % 20.0 - 40.0 Flow Cytometry /
Microscopy
MONOCYTES 11.6 % 2.0 - 10.0 Flow Cytometry /
Microscopy
EOSINOPHILS 2.0 % 1.0 - 6.0 Flow Cytometry /
Microscopy
BASOPHILS 00 % <=2 Flow Cytometry /
Microscopy
Absolute Leucocyte Count
ABSOLUTE NEUTROPHIL COUNT 2.75 Thousand / cmm 2.0 - 7.0 Flow Cytometry /
Microscopy
ABSOLUTE LYMPHOCYTE COUNT 2.76 Thousand/cmm 1.0 - 3.0 Flow Cytometry /
Microscopy
ABSOLUTE MONOCYTE COUNT 0.74 Thousand/cmm 0.200-1.00 Flow Cytometry /
Microscopy
ABSOLUTE EOSINOPHIL COUNT, Whole Blood 0.13 Thousand/cmm 0.02 - 0.5 Flow Cytometry /
EDTA Microscopy
NEUTROPHIL/LYMPHOCYTE RATIO (N/L 1.00 1-3 Calculated
RATIO)
RED BLOOD CELL COUNT 4.09 mill./cmm 3.8 - 4.8 DC Detection
PACKED CELL VOLUME(PCV-HEMATOCRIT) 34.90 % 36.0-46.0 RBC Pulse Height
Detection
MEAN CELL VOLUME(MCV) 85.30 fl 83.0 - 101.0 DC Detection /Calculation
MEAN CELL HAEMOGLOBIN 31.80 g/dL 31.5 - 34.5 Calculation
CONCENTRATION (MCHC)
MEAN CELL HAEMOGLOBIN(MCH) 27.1 pg. 27.0 - 32.0 DC Dectection
Calculation
RED CELL DISTRIBUTION WIDTH (RDW) 15.6 % 11.6 - 14.0 Calculated
ERYTHROCYTE SEDIMENTATION RATE (ESR), 24.00 mm/1st Hr <=20 Westergrens
Sodium Citrate

Draft By: Shekhar Page 1 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:48AM
LAB No : PN160759 Reported : 04/Oct/2024 11:50AM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

DEPARTMENT OF HAEMATOLOGY
Investigation Name Result Unit Bio. Ref. Range Method

PLATELET COUNT, Whole Blood EDTA 242.0 thousand/cmm 150.0-410.0 Hydrodynamically

Focused Impedence
MEAN PLATELET VOLUME;MPV 11.30 fl 7.8-11.2 DC Detection/Caluclated

Draft By: Shekhar Page 2 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:48AM
LAB No : PN160759 Reported : 04/Oct/2024 12:49PM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

DEPARTMENT OF CLINICAL BIOCHEMISTRY

Investigation Name Result Unit Bio. Ref. Range Method

GLYCOSYLATED HB (HBA1C), Whole Blood 9.70 % 0.0 - 5.7 Immunoturbidimetry

(EDTA)
ESTIMATED AVERAGE GLUCOSE(EAG) 231.69 mg/dL
Interpretation:
Expected Values Normal < 5.7 %
Pre diabetes 5.7 - 6.4 %
Diabetes 6.5 % or higher.
Treatment Goal <7%

LIPID PROFILE
CHOLESTEROL, Serum 190 mg/dL < 200.0 CHOD-POD
HDL CHOLESTEROL, Serum 51.00 mg/dL 40.0 - 60.0 Homogenous Enzymatic
Colorimetric Assay
LDL CHOLESTEROL, SERUM 115 mg/dL 0.0 - 100.0 Homogeneous Enzymatic
Colorimetric Assay
TRIGLYCERIDE, Serum 154 mg/dL < 150.0 Enzymatic colorimetric
VLDL CHOLESTEROL 30.86 mg/dL 10.0 - 50.0 Calculation
NON-HDL CHOLESTEROL 140 mg/dL < 130 Calculation

25 HYDROXY VITAMIN D, Serum 158.6 nmol/L 81-250 ECLIA

VITAMIN B12, Serum 364.40 pg/mL 197.0 - 771 ECLIA

TSH, Serum 13.010 µIU/ml 0.27-4.2 ECLIA

Pregnancy: Ist Trimester IInd trimester IIIrd trimester

BRI (uIU/ml) 0.1- 2.5 0.2-3.0 0.3-3.0

LFT + KFT
BILIRUBIN(TOTAL), Serum 0.403 mg/dL 0-1.1 Diazo
BILIRUBIN (CONJUGATED), SERUM 0.187 mg/dL 0.0 - 0.2 Diazo
BILIRUBIN (UNCONJUGATED), SERUM 0.216 mg/dL 0.10 - 1.0 Calculation

Draft By: Rupali Page 3 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:48AM
LAB No : PN160759 Reported : 04/Oct/2024 12:49PM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

DEPARTMENT OF CLINICAL BIOCHEMISTRY

Investigation Name Result Unit Bio. Ref. Range Method

SGOT (AST), Serum 28.40 U/L <32 IFCC without P5P

SGPT (ALT), Serum 41.70 U/L <33 IFCC without P5P
ALKALINE PHOSPHATASE, Serum 75 U/L 35 - 104 IFCC, P- Nitrophenyl
Phosphate
PROTEIN TOTAL, Serum 7.18 g/dL 6.6 - 8.7 Biuret
ALBUMIN, Serum 4.32 g/dL 3.5-5.2 BCG
GLOBULIN 2.86 g/dl 2-3.5 Calculation
A/G RATIO 1.51 :1 0.8-2.00 Calculation
UREA, Serum 26.10 mg/dL 19-47 Kinetic Urease
BLOOD UREA NITROGEN, Serum 12.2 mg/dL 8.0 - 23.0 Kinetic Urease
CREATININE, Serum 1.04 mg/dL 0.60-1.20 Jaffe
EGFR, SERUM 60.7 ml/min/1.73m² Calculation
URIC ACID, Serum 6.00 mg/dL 2.4-5.7 Uricase
SODIUM, Serum 136 mmol/L 136-145 ISE direct
POTASSIUM, Serum 4.6 mmo/L 3.5-5.1 ISE Direct
CHLORIDE, Serum 103.0 mmol/L 98-107 ISE Direct
CALCIUM, Serum 9.57 mg/dL 8.8-10.2 NM - BAPTA
PHOSPHOROUS, Serum 3.4 mg/dL 2.5 - 4.5 Phosphomolybdate UV

Draft By: Rupali Page 4 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:48AM
LAB No : PN160759 Reported : 04/Oct/2024 12:49PM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

Investigation Name Result Unit Bio. Ref. Range Method

Test Interpretations
Glycosylated Hemoglobin (HbA1c)
Comments
Glycosylated Haemoglobin (HbA1c) is a measure of long term (2-3 months) glycemic control. HbA1c values 5.7 -6.4 %
indicates increased risk for Diabetes. HbA1c values >6.5 % has been included in the latest guidelines for the diagnosis of
Diabetes. It helps in more effective monitoring of blood glucose level to prevent diabetic complications. It is recommended
that HbA1c test should be performed twice a year in patients who are meeting treatment goals and quarterly in patients
who are not meeting glycemic control.

eAG is a new term recommended by ADA (American Diabetes Association) in diabetes management, by which HbA1c results
can be reported to the patients using the same units (mg/dL or mmol/L) that patients see routinely in blood glucose
measurements. One advantage of using eAG as a measure of glucose control is that it will help patients more directly see
the difference between their individual meter readings and how they are doing with their glucose management overall, but
the values of eAG is unlikely to match the average glucose level shown on a person's meter, because people with diabetes
are more likely to test more often when their blood glucose levels are low as in fasting and before meals, but eAG
represents an average of their glucose levels 24 hrs. a day, including post meal periods of higher blood glucose when
people are less likely to test. Also some diabetologists in UK do not like to report eAG

Lipid Profile

Interpretation Total cholesterol LDL Triglyceride Non HDL Cholesterol in

(mg/dL) Cholesterol(mg/dL) (mg/dL) mg/dL
Optimal < 200 < 100 < 150 <130
Above Optimal - 100 - 129 - 130-159
Borderline High
200 - 239 130-159 150 - 199 160-189

High > 239 160 - 189 200 - 499 190-219

Very High - > 189 > 449 >=220

Interpretation :

Testing of Lipid Profile helps physicians and their patients take a more proactive and personalised approach to
cardiovascular risk.

Indians are at a greater risk of atherosclerotic cardiovascular disease (ASCVD) and also at an earlier age as compared to
western population. There are many correctable risk factors for ASCVD and dyslipidemia is the most important of these.

Draft By: Rupali Page 5 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:48AM
LAB No : PN160759 Reported : 04/Oct/2024 12:49PM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

Investigation Name Result Unit Bio. Ref. Range Method

Other important high risk factors are smoking , Diabetes , sedentary lifestyle, hypertension , family history,low HDL-C etc.

LDL-C should be the primary target for therapy, lowering it helps to achieve desired reduction in risk of ASCVD.

Low HDL-C is an independent risk factor for ASCVD.

Non HDL cholesterol is a stronger predictor of CVD as it measures all atherogenic lipoprotein including LDL & TG rich
lipoprotein remnant.

Elevated TG is ssociated with increased risk of ASCVD independent of LDL-C levels. A combination of high TG and LD-
C imparts even greater risk.

Note:
Lipid Association of India (LIA) does not find any advantage in permorming lipid profile in a fasting state. In most patients, there is
usually a clinically unimportant increase in TG concentration by 18-36 mg/dL on average 2-6 hrs after eating a normal meal.
Fasting lipid rofile are indicated if;

Non fasting TG level > 400 mg/dL

Familial dyslipedemia
Followup patient with hypertriglycerides

Vitamin D ( 25 Hydroxy)
Interpretation:

Levels Clinical Implication

Severe deficiency < 25 nmol/L Could be associated with osteomalacia or rickets.
Mild or moderate May be associated with increased risk of osteoporosis or
25-80 nmol/L
deficiency secondary hyperparathyroidism
81-250
Optimum levels Optimum levels in the normal population.
nmol/L
250 nmol/L is the lowest reported level associated with
toxicity in patients without primary hyperparathyroidism who
have normal renal function. Most patients with toxicity have
Possible toxicity > 250 nmol/L
levels > 375 nmol/L. patients with renal failure can have very
high 25 - OH Vit D levels without any signs of toxicity.

Draft By: Rupali Page 6 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:48AM
LAB No : PN160759 Reported : 04/Oct/2024 12:49PM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

Investigation Name Result Unit Bio. Ref. Range Method

These Reference ranges represent clinical decision values that apply to males and females of all ages, rather than population based
reference values.

Reference :
NIH clinical center USA & US National osteoporosis foundation.
Vitamin B12 (Methylcobalamin)
Comments

Vitamin B12 (cobalamin) is necessary for hematopoiesis and normal neuronal function. In humans, it is obtained only from
animal proteins and requires intrinsic factor (IF) for absorption. Vitamin B12 deficiency may be due to lack of IF secretion
by gastric mucosa (eg, gastrectomy, gastric atrophy) or intestinal malabsorption (eg, ileal resection, small intestinal diseases).
Vitamin B12 deficiency frequently causes macrocytic anemia, glossitis, peripheral neuropathy, weakness, hyperreflexia,
ataxia, loss of proprioception, poor coordination and affective behavioral changes.
Pernicious anemia is a macrocytic anemia caused by vitamin B12 deficiency that is due to a lack of IF secretion by gastric
mucosa.
Vitamin B12 concentrations <150 ng/L are considered evidence of vitamin B12 deficiency. Serum methylmalonic acid and
homocysteine levels are also elevated in vitamin B12 deficiency states.
Other conditions are also known to decrease the serum vitamin B12 concentration: pregnancy, drugs such as
aspirin,anticonvulsants, colchicines, ethanol ingestion, contraceptive hormones and smoking.

TSH
Comments
Thyroid stimulating hormone ( TSH ) is an important marker of thyroid function in our body . The prime function of TSH is to
regulate the synthesis and secretion of the thyroid hormones viz. T3 and T4. The determination of TSH serves as the initial
test in thyroid diagnostics. Alteration in the TSH level indicates either hyperthyroidism ( low TSH level ) or hypothyroidism
(high TSH). However, only TSH determination would not help in disease diagnosis. It should always be performed along with
Free T3 and Free T4 for a proper clinical diagnosis. Various research studies have indicated considerable levels of biological
and analytical variations in TSH measurement. It may be attributed to mainly due to the pulsatile secretion of the hormone
and fairly short half life ( 1 - 2 hrs). Research studies have indicated that circadian variation (i.e variation of morning and
evening samples) of as high as 50% variation. This is more so even in case of pregnant ladies. Hence TSH interpretation
should be both diagnostic and clinical, not a single factor alone.

Draft By: Rupali Page 7 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:42AM
LAB No : PN160759 Reported : 04/Oct/2024 05:18PM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

DEPARTMENT OF EPS

NERVE CONDUCTION STUDIES UPPER LIMBS

MOTOR AND SENSORY NERVE CONDUCTION

CLINICAL INDICATION / HISTORY :- PAIN IN BOTH HANDS

PREVOIUS REPORT:- No
OBSERVATIONS:-

Amplitude of both median and ulnar nerves are normal.

Amplitude of both biceps and deltoid nerves are normal.

Distal latencies of both median motor are prolonged and conduction velocities are normal.

F-waves latencies of both median and ulnar nerves are normal.

Both median sensory amplitude are normal and conduction velocities are reduced.

Both radial and ulnar nerves sensory are normal.

IMPRESSION:
THE ELECTROPHYSIOLOGY STUDY SHOWS MILD TO MODERATE BILATERAL
CARPAL TUNNEL SYNDROME.
To correlate clinically.

Draft By: Dinesh Sareen Page 8 of 9

 Patient Name : Mrs.RASHMI TRIPATHI Reg. Date : 04/Oct/2024 08:42AM
Age/Gender : 61 Y 5 M 22 D /F ,DOB:- 13-Apr-1963 Collected : 04/Oct/2024 08:42AM
LAB No : PN160759 Reported : 04/Oct/2024 05:18PM
Mobile No : 9891910400 Status : Final Report
Refer Doctor :Dr.CGHS :
: :

DEPARTMENT OF EPS

*** End Of Report ***

Draft By: Dinesh Sareen Page 9 of 9