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Insect Metamorphosis Presentation

Insect development involves a process called metamorphosis where the insect undergoes dramatic physical changes in form and structure. There are three main patterns of insect metamorphosis: ametabolous (no change in form), hemimetabolous (gradual changes through nymph stages), and holometabolous (complete transformation from larva to pupa to adult). This process is controlled by two hormones - juvenile hormone which maintains the juvenile state, and ecdysone which triggers molting and metamorphosis. The balance and timing of these hormones determines whether the insect remains in the same form or progresses to a new developmental stage.

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173 views10 pages

Insect Metamorphosis Presentation

Insect development involves a process called metamorphosis where the insect undergoes dramatic physical changes in form and structure. There are three main patterns of insect metamorphosis: ametabolous (no change in form), hemimetabolous (gradual changes through nymph stages), and holometabolous (complete transformation from larva to pupa to adult). This process is controlled by two hormones - juvenile hormone which maintains the juvenile state, and ecdysone which triggers molting and metamorphosis. The balance and timing of these hormones determines whether the insect remains in the same form or progresses to a new developmental stage.

Uploaded by

Tomi Oboh
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© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
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METAMORPHOSIS OF INSECT

INTRODUCTION

• Metamorphosis refers to the way that certain


organisms develop, grow, and change form.
• Dalam proses pertumbuhan tersebut terjadi
proses pergantian kulit yang dikenal dengan
istilah ecdysis atau molting dan sisa kulit yang
terkelupas disebut exuviae.
• Selama pertumbuhan berlangsung akan
mengalami bebrapa kali pergantian kulit dan
bentuk serangga antara dua masa pergantian
kulit tersebut disebut instar
• Insect molting and metamorphosis are
controlled by two effector hormones: the
steroid 20-hydroxyecdysone (20E) and the
lipid Juvenile Hormone (JH)
There are Three Major Patterns of
Insect Development
Ametabolous
• Have a pronymph stage immediately after hatching
• Unchanged in form except their size

Hemimetabolous
• After pronymph, the insect looks like an immature adult called nymph
• The rudiments of the wings, genital organs, and other adult structures are
present. The rudiments of the wings, genital organs, and other adult
structures are present

Holometabolous
• There is no pronymph stage
• The juvenile form that hatches from the egg is called a larva
Figure.
Modes of insect development. Molts are
represented as arrows. (A) Ametabolous
(direct) development in a silverfish. After a
brief pronymph stage, the insect looks like a
small adult. (B) Hemimetabolous (gradual)
metamorphosis in a cockroach. After a very
brief pronymph phase, the insect becomes a
nymph. After each molt, the next nymphal
instar looks more like an adult, gradually
growing wings and genital organs. (C)
Holometabolous (complete) metamorphosis in
a moth. After hatching as a larva, the insect
undergoes successive larval molts until a
metamorphic molt causes it to enter the pupal
stage. Then an imaginal molt turns it into an
adult.
Figure .
Regulation of insect metamorphosis. (A) Structures of juvenile hormone, ecdysone, and the active molting
hormone 20-hydroxyecdysone. (B) General pathway of insect metamorphosis. Ecdysone and juvenile
hormone together cause molts to keep the status quo and form another larval instar. When there is a
lower concentration of juvenile hormone, the ecdysone-induced molt produces a pupa. When ecdysone
acts in the absence of juvenile hormone, the imaginal discs differentiate, and the molt gives rise to the
adult.
Figure. Juvenile hormone (JH) and 20-hydroxyecdysone (20E) titers during Drosophila development [64]. The
balance between 20E (denoted by the solid line) and JH (denoted by the broken line) determines the normal
course of insect development.
The Molecular Biology of Hydroxyecdysone Activity

Ecdysone Receptors

Figure. Formation of the ecdysone receptors. Alternative mRNA splicing of the ecdysone
receptor (EcR) transcript creates three types of EcR mRNAs. These generate proteins
having the same DNA-binding site (blue) and hydroxyecdysone-binding site (red), but
with very different amino termini.
Binding of 20-hydroxyecdysone to DNA

Figure.
Hydroxyecdysone-induced puffs in cultured salivary
gland cells of D. melanogaster. The chromosome
region here is the same as in Figure 4.13. (A)
Uninduced control. (B-E) Hydroxyecdysone-
stimulated chromosomes at (B) 25 minutes, (C) 1
hour, (D) 2 hours, and (E) 4 hours. (Photographs
courtesy of M. Ashburner.)
Figure 18.24
The Ashburner model of hydroxyecdysone regulation of transcription. Hydroxyecdysone binds to its receptor, and this
compound binds to an early puff gene and a late puff gene. The early puff gene is activated, and its protein product (1)
represses the transcription of its own gene and (2) activates the late puff gene, perhaps by displacing the ecdysone
receptor.

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