Patient : Ms POONAM Lab Ref. No.

: 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

.
COMPLETE BLOOD COUNT
HAEMOGLOBIN 13.60 gm/dl 12-17
Methodology:Colorimetric

TOTAL LEUCOCYTE COUNT 10300 /cumm 4000-11000

Methodology: Electro Impedance

DIFFERENTIAL LEUCOCYTE COUNT

Neutrophils 71.10 %. 40-75
Methodology: Manual

Lymphocytes 22.70 %. 20-45

Methodology: Manual

Eosinophil 2.48 % 00-06

Methodology: Manual

Monocyte 3.72 % 00-08

Methodology: Manual

Basophils 00 % 00-01
PLATELET COUNT 2.64 lacs/mm3 1.50 -4.50
Methodology: Electro Impedance

TOTAL R.B.C. COUNT 4.35 million/cumm 3.5-6.5

Methodology: Electro Impedance

P.C.V./ Haematocrit value 39.70 % 35-54

Methodology: Calculated

MCV 91.26 fL 76-96

Methodology: Calculated

MCH 31.26 pg 26.00-34.00

Methodology: Calculated

MCHC 34.26 g/dl 30.50-35.50

Methodology: Calculated

RDW-CV 13.10 % 10-16

Methodology: Calculated

MPV 11.90 u3 6.0-11

Methodology: Calculated

P-LCR 41.30 % 10.0-60.0

Methodology: Calculated

P-LCC 109.00 % 10.0-99.0

Page 1 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

Methodology: Calculated

LIVER PROFILE
SERUM BILIRUBIN
TOTAL 0.52 mg/dL 0.3-1.0
Methodology: Diazo

DIRECT 0.32 mg/dL 0.2-0.6

Methodology: Diazo

INDIRECT 0.20 mg/dL 0.1-0.4

Methodology: Calculated

SERUM PROTEINS
Total Proteins 6.54 Gm/dL 5.5-8.5
Methodology: Biuret

Albumin 3.52 Gm/dL 3.5 - 5.5

Methodology: BCG

Globulin 3.02 Gm/dL 2.3 - 3.5

Methodology: Calculated

A : G Ratio 1.17 0.0-2.0

Methodology: Calculated

SGOT 17.00 IU/L 0-40

Methodology: IFCC

SGPT 12.20 IU/L 0-40

Methodology: IFCC

SERUM ALK.PHOSPHATASE 110.00 IU/L 00-200

Methodology: AMP

NORMAL RANGE : BILIRUBIN TOTAL

Premature infants. 0 to 1 day: <8 mg/dL Premature infants. 1 to 2 days: <12 mg/dL Adults: 0.3-1 mg/dL.
Premature infants. 3 to 5 days: <16 mg/dL Neonates, 0 to 1 day: 1.4-8.7 mg/dL
Neonates, 1 to 2 days: 3.4-11.5 mg/dL Neonates, 3 to 5 days: 1.5-12 mg/dL Children 6 days to 18 years: 0.3-1.2 mg/dL
COMMENTS--
Total and direct bilirubin determination in serum is used for the diagnosis,differentiation and follow -up of jaundice.Elevation of SGPT is found in
liver and kidney diseases such as infectious or toxic hepatitis,IM and cirrhosis.Organs rich in SGOT are heart ,liver and skeletal muscles. When any
of these organs are damaged,the serum SGOT level rises in proportion to the severity of
RENAL PROFILE
BLOOD UREA 27.80 mg/dL. 10-45
Methodology: Urease UV

SERUM CREATININE 0.67 mg/dL. 0.5-1.5

Page 2 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

Methodology: Jaffe's

SERUM URIC ACID 6.2 mg/dL. 3.5 - 7.2

Methodology: Urease-POD

SERUM SODIUM (Na) 142 m Eq/litre. 135 - 155

Methodology: ISE

SERUM POTASSIUM (K) 4.3 m Eq/litre. 3.5 - 5.5

Methodology: ISE

SERUM CHLORIDE (Cl) 102 mmol/L 96-110

Methodology: ISE

INTERPRETATION----
Urea is the end product of protein metabolism.It reflects on funcioning of the kidney in the body. Creatinine is the end product of creatine
metabolism.It is a measure of renal function and eleveted levels are observed in patients typically with 50% or greater impairment of renal
function.Sodium is critical in maintaining water & osmotic equilibrium in extracellular fluids.Disturbances in acid base and water balance are
typically reflected in the sodium concentrations .Potassium is an essential element involved in critical cell functions. Potassium levels are
influenced by electrolyte intake ,excretion and other means of elemination ,exercise ,hydration and medications. Calcium imbalance my cause a
spectrum of disease . High concentrations are seen in Hyperparathyroidism,Malignancy & Sarcoidosis. Low
LIPID PROFILE,EXT
CHOLESTEROL-TOTAL 144.00 mg/dl 00-200
Methodology: CHOD-PAP
TRIGLYCERIDES 132.60 mg/dl 00-150
Methodology: GPO
HDL CHOLESTEROL 65.00 mg/dl 30-80
Methodology: Enzymatic
LDL CHOLESTEROL 26.52 mg/dl 00-130
Methodology: Calculated
VLDL CHOLESTEROL 52.48 mg/dl 15-40
Methodology: Calculated
TOTAL CHO/ HDL CHO RATIO 2.22
Methodology: Calculated
LDL / HDL CHOLESTEROL RATIO 26.52
Methodology: Calculated
APOLIPOPROTEIN A (APO-A) 1.52 g/L 1.04 - 2.02
Methodology: Immunoturbidimetry
APOLIPOPROTEIN B (APO - B),Serum 0.89 g/L 0.66 - 1.33
Methodology: Immunoturbidimetry

Page 3 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

CHOLESTEROL LDL- CHOLESTEROL CHO/HDL RATIO

Acceptadle/Low Risk : <200 mg/dL : <130 mg/dL <4.5

Borderliune High Risk : 200 - 239 mg/dL 130-59 mg/dL 4.5 -6.0
High Risk : <240 mg/dL : <160 mg/dL <6.0

ALERT!!! 10-12 hours fasting is mandatory for lipid parameters.If not,values might fluctuate.
CLINICAL NOTES
Lipid profile is initial screening tool for abnormalities in lipids. The results of this test can identify certain genetic diseases & can
determine approximate risks for
cardiovascular disease, certain forms of pancreatitis. Hypertriglyceridemia is indicative of insulin resistance when present with low
HDL & elevated LDL, while elevated TG
is risk factor for coronary artery disease,especially when low HDL is present.TG of 500mg/dL or more can be concerning for
development of pancreatitis.
BLOOD SUGAR RANDOM 98 mg/dl 80 - 140
Methodology: GOD-POD

SERUM CALCIUM 9.40 mg/dl 8.5 - 10.5

Methodology: Arsenazo

Triiodothyronine (T3) 1.48 ng/ml 0.60-1.81

Thyroxine (T4) 9.00 ug/dl 3.5-12.6
THYROID STIMULATING HORMONE [TSH.] 2.12 mIU/L 0.35-5.50

Kindly correlate clinically.

INTERPRETATION
1. Primary hyperthyroidism is accompanied by elevated serum T3 & T4 values along with depressed TSH level.
2. Primary hypothyroidism is accompanied by depressed serum T3 and T4 values & elevated serum TSH levels.
3. Normal T4 levels accompanied by high T3 levels and low TSH are seen in patients with T3 thyrotoxicosis.
4. Normal or low T3 & high T4 levels indicate T4 thyrotoxicosis ( problem is conversion of T4 to T3)
5. Normal T3 & T4 along with low TSH indicate mild / subclinical HYPERTHYROIDISM .
6. Normal T3 & low T4 along with high TSH is seen in HYPOTHYROIDISM .
7. Normal T3 & T4 levels with high TSH indicate Mild / Subclinical HYPOTHYROIDISM .
8. Slightly elevated T3 levels may be found in pregnancy and in estrogen therapy while depressed levels may be encountered in severe
illness , malnutrition , renal failure and during therapy with drugs like propanolol.
9. Although elevated TSH levels are nearly always indicative of primary hypothyroidism . rarely they can result from TSH secreting pituitary
tumors (secondary hyperthyroidism.

Vitamin D (25 - Hydroxy) 42.00 ng/ml 30-100

Page 4 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

REFERENCE RANGE:
DEFICIENCY : <20 ng/mL
INSUFFICIENCY : 20-30 ng/ml
SUFFICIENCY : 30-100 ng/ml
INTOXICITY : >100ng/mL

COMMENTS:Lower-than-normal levels suggest a vitamin D deficiency. This condition can result from Lack of exposure to sunlight ,Lack of
adequate vitamin D in the diet, Liver and kidney diseases and Malabsorption. A vitamin D deficiency may lead to:
*Low blood calcium levels (hypocalcaemia)
*Thin or weak bones (rickets, osteoporosis and osteomalacia)
*High levels of parathyroid hormone (secondary hyperparathyroidism) Total 25-hydroxyvitamin D (D2 + D3) is the correct measure of Vitamin D
status.Higher-than-normal levels suggest excess vitamin D, a condition called hypervitaminosis D. It is usually caused by vitamin D in the form of
doctor -prescribed dietary supplements.
95% of ser
SERUM VITAMIN B 12 420.00 pg/ml 211 - 911

Vitimin B 12 and folate are critical to normal DNA synthsis, which in turn affects erythrocyte maturation.
Vitamin B 12 is also necessary for myelin sheat formation and maintenance. Pernicious anemia is a macrocytic anemia
caused by vitamin B12 deficiancy that is due to lack of intrinsic factor. Low vitamin B12 intake, gastrectomy, diseases of small
intestine, malabsorption and trans-cobalamine deficiency can also cause vitamin B12 deficiancy.

METHOD : CLIA
AUSTRALIA ANTIGEN (HBsAG) NON REACTIVE AI

Interpretation :
:Thisassay detects the first serological marker of Hepatitis B as early as 4-16 weeks after exposure.
It persists during acute illness and disappears 12-20 week after onset of symptoms.
The titres during the period of viral replication and is frequently associated with infectivity.
Persistence of HBsAg for more than 6months indicates development of carrier state or chronic liver disease.
Uses
* Routine screening of blood and blood products to prevent prevent transmission of Hepatitis B virus (HBS)
to recipients
* To diagnose suspected HBV infection and monitor the status of infected infected individuals
* To evaluat
HEPATITIS C VIRUS NON REACTIVE NEGATIVE

Page 5 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

Comments:

HEPATITIS C VIRUS accounts for about 95% of hepatitis infections in recipients of blood transfusion and 50% of cases of Sporadic nanb
hepatitis.
HCV commonly gives origin to asymptomatic hepatitis and chronicity develop in a high number of cases, sometime evolving in severs forms of
illness, as hepatocarcinoma. IGM Antibodies directed to the major immunodominant patient determinants of the viral proteins are detected in
patients infected with HCV, early in the couses of infection and in patients upon reactivation of viral relocation in Hepatocites.

HIV 1 &2 NON REACTIVE NEGATIVE

METHOD RAPID CARD

COMMENTS: Non reactive results indicate that antibodies to HIV I/II have not been detected in
the sample. This implies either non exposure to HIV I/II or that the individual might be in the
`Window Period`, that is, prior to the development of detectable level of Antibodies. Therefore, a
non-reactive result does not exclude the possibility of a persisting HIV infection. Reactive
samples must be confirmed by using HIV Western Blot as some degree of cross reactivity for HIV
antibodies has been noted with certain other naturally occurring antibodies and also antibodies
formed in response to other bacterial and viral infections , and in certain autoimmune disorders.
Post test counseling will be done by the concerned referring doctor. The sensitivity and
specificity of this test has been determined by National HIV Reference Centers of Govt .of India
and WHO collaborating cent
R.A. FACTOR NEGATIVE NEGATIVE

NOTE:-
Gives useful objective evidence of RA, but a negative test does not rule out RA.
Negative in a third of patients with definite RA.
Positive result in < 50% during first 6 months of disease.
Sensitivity : 50 -75% Specificity :75 - 90%.

C-REACTIVE PROTEIN(QUALITATIVE) NEGATIVE NEGATIVE

CLINICAL SIGNIFICANCE.
CRP is an acute-phase protein present in normal serum, which increases significantly after most forms of
tissue injuries,bacterial and virus infection , inflammation and malignant neoplasia. During tissue necrosis and
inflammation resulting from microbial infections.
GLYCOSYLATED HAEMOGLOBIN 5.0 % 4.5-6.5

Page 6 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

Methodlogy:HPLC

EXPECTED RESULTS :
------------------
Diagnosing diabetes American Diabetes Association (ADA)
-Hemoglobin A1c (HbA1c) >6.5%
Therapeutic goals for glycemic control (ADA)
- Goal of therapy: <7.0% HbA1c
- Action suggested: >8.0% HbA1c The glycosylated hemoglobin assay has been validated as a reliable indicator of mean blood glucose levels for
a period of 8-12 week period prior to HBA1C determination.ADA recommends the testing twice a year in patients with stable blood glucose, and
quarterly, if treatment changes, or if blood glucose levels are unstable.
METHOD :HPLC
SERUM FOLIC ACID 8.30 ng/ml

NORMAL RANGE.
0.35 - 3.37 : Deficient
3.38 - 5.38 : Indeterminate
> 5.38 : Normal
Folate deficiency causes megaloblastic anemia and eventually leukopenia and thrombocytopenia. Folic acid is believed to play a role in birth
defects such as spina bifida, anencephaly, and oro-facial clefts as well as reducing cardiovascular morbidity and mortality. Symptoms of deficiency
take about 3 months to appear and can be caused by inadequate intake, increased body demand, or folate antagonism by drugs.For diagnostic
purposes, the Folate
findings should always be assessed in conjunction with the patient`s medical history, clinical examination and other findings.

PROTHROMBIN TIME
On Patients Blood 18.0 Seconds 0-21.00
Prothrombin Control 13.0
International Normalized Ratio (INR) 1.4

Page 7 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

NOTE: 1.INR is the parameter of choice in monitoring adequacy of oral anticoagulant therapy.Appropriate therapeutic range varies with disease
and treatment intensity.
2.Prolonged INR suggests potential bleeding disorder/bleeding complications.
3.Results should be clinically correlated.
4.Test conducted on Citrated plasma.
Recommended Therapeutic range for Oral Anticoagulant therapy
INR 2.0-3.0
* Treatment of Venous thrombosis and pulmoonary embolism
* Prophylaxis of Venous thrombosis (High risk surgery)
* Prevention of systemic embolism in tissue heart valves,AMI, Valvular heart disease & Atrial
fibrillation
* Bileaflet mechanical valve in aortic position.
INR 2.5-3.5 :
* Mechanical prosthetic val
A.P.T.T 33.0 25-50

The activated partial thromboplastin time (aPTT, often called PTT) test is a measure of the functionality of the intrinsic andcommon pathways of
the coagulation cascade. The body uses the coagulation cascade to produce blood clots to seal off injuries to blood vessels and tissues, to prevent
further blood loss, and to give the damaged areas time to heal. The cascadeconsists of a group of coagulation factors. These proteins are activated
sequentially along either the extrinsic (tissue related)or intrinsic (blood vessel related) pathways. The branches of the pathway then come together
into the common pathway, andcomplete their task with the formation of a stable blood clot. When a person starts bleeding, these three pathways
have towork together.
Each component of the coagulation cascade must be functioning properly
SERUM AMYLASE (ALFA) 52.0 U/litre. 30-118
Methodology: IFCC

DIAGNOSTIC SIGNIFICANCE : Serum amylase is elevated in acute pancreatitis.

-In acute pancreatitis alfa amylsae starts rising approximatelyfour hours after
the onset of pain, reaches peak at 24 hours and remains elevated for 3to7days.
-High levels of amylase are also associated with other disorders, like biliary
tract diseases, severe glomerular dysfunction and salivary gland disorders.

SERUM LIPASE 25.0 IU/L 6-51

Methodology: IFCC

BLEEDING TIME 1.18 Min./sec. 01-05

PROLACTIN 9.10 ng/ml 2.1-17.7

Page 8 of 9
Patient : Ms POONAM Lab Ref. No. : 164
AgeSex : 37 Year FEMALE Registered On. : 13-Dec-2024
Card/Ins. No. : 05070390593290 Collected On. : 13-Dec-2024

Referd By. : Self Tested On. : 13-Dec-2024

Sample : Printed On. : 22-Dec-2024

Test Name Test Value Unit BIOLOGICAL REF-RANGE

INTERPRETATION:
Non Pregnant Female 2.8-29.2 ng/ml
Pregnancy 9.7-208.5 ng/ml
Post-Menopausal 1.8-20.3 ng/ml
Male 2.1-17.7 ng/ml
-The major physiologic action of prolactin is the initiation and maintenance of lactation in women.
-Hyperprolactinemia has been established as a common cause of infertility and gonadal disorders in men and women.
-Causes of increased prolactin concentrations include pituitary tumours, amenorrhoea and/or galactorrhoea, primary
hypothyroidism, anorexia nervosa, polycystic ovarian syndrome, renal failure and ectopic production. Women taking oral
contraceptives or receiving estrogen therapy can have elevated prolactin concentration. Stress, coitus, some psychotropic
and antihypertensive drugs may give falsely elevated values.
-Causes of decreased prolactin concentrations include hypopituitarism, post partum, administration of certain drugs like Ldopa, apomorphine,
clonidine and bromocriptine.
LIMITATIONS:
-Prolactin levels have been found to be influenced by various factors other than the diseased state.
-Prolactin may exist in alternate structural forms (e.g. macroprolactin) which may exhibit variable levels of physiological
activity. Additional information may be required for diagnosis.
-Specimens from patients who have received preparations of mouse monoclonal antibodies for diagnosis or therapy may
show either falsely elevated or depressed values.
SERUM IgE 30.00 IU/mL <190

COMMENT:Because IgE is a mediator of the allergic response, quantitative measurement of serum IgE, when
integrated with other clinical indicators, can provide useful information for the differential clinical diagnosis of atopic and
not-atopic disease. Patients with atopic disease, including allergic asthma, allergic rhinitis, and atopic dermatitis
commonly have moderately elevated serum IgE levels. However, a serum IgE level which is within the range of normally
expected values does not rule out a limited set of IgE-dependent allergies. Total serum IgE levels may also be elevated
in the presence of some clinical conditions that are not related to allergy. Thes
PARATHYROID HORMONE (P.T.H.) 23.00 pg/mL 12-88

Comment:
PTH levels provide useful aid in differential diagnosis of hypercalcemia.
It is not intended for the diagnosis or management of malignancy.
It is important that PTH levels are interpreted in light of calcium levels along with other clinical findings.
Method : Chemiluminescence.

Mobile

--------------End of Report-------------

Page 9 of 9