File 6
File 6
HAEMATOLOGY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Comment:
As per the recommendation of International council for Standardization in Hematology, the differential leucocyte counts are
additionally being reported as absolute numbers of each cell in per unit volume of blood.
DHSS : Double Hydrodynamic Sequential System Flowcytometry
Calculated parameters are either derived from Impedence measure, RBC pulse measurement, RBC/platelet histograms or
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PO No :PO3758436865-720
HAEMATOLOGY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
formula derived.
Comment:
ESR provides an index of progress of the disease and is widely used as an indicator of inflammation, infection, trauma, or
malignant diseases. Changes are more significant than a single abnormal test
It is specifically indicated to monitor the course or response to the treatment of diseases like rheumatoid arthritis,
tuberculosis bacterial endocarditis ,acute rheumatic fever ,Hodgkins disease,temporal arthritis , and systemic lupus
erythematosis; and to diagnose and monitor giant cell arteritis and polymyalgia rheumatica.
An elevated ESR may also be associated with many other conditions, including autoimmune disease, anemia,
infection,malignancy,pregnancy, multiple myeloma, menstruation, and hypothyroidism.
Although a normal ESR cannot be taken to exclude the presence of organic disease, its rate is dependent on various
physiologic and pathologic factors.
The most important component influencing ESR is the composition of plasma. High level of C-Reactive Protein, fibrinogen,
haptoglobin, alpha-1antitrypsin, ceruloplasmin and immunoglobulins causes the elevation of Erythrocyte Sedimentation
Rate.
Drugs that may cause increase ESR levels include: dextran, methyldopa, oral contraceptives, penicillamine, procainamide,
theophylline, and Vitamin A. Drugs that may cause decrease levels include: aspirin, cortisone, and quinine
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PO No :PO3758436865-720
HAEMATOLOGY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Comment:
Interpretation: HbA1c%
≤5.6 Normal
5.7-6.4 At Risk For Diabetes
≥6.5 Diabetes
Comments:
A 3 to 6 monthly monitoring is recommended in diabetics. People with diabetes should get the test done more often if their blood
sugar stays too high or if their healthcare provider makes any change in the treatment plan. HbA1c concentration represent the
integrated values for blood glucose over the preceding 8-12 weeks and is not affected by daily glucose fluctuation, exercise &
recent food intake.
Please note, Glycemic goal should be individualized based on duration of diabetes, age/life expectancy, comorbid conditions,
known CVD or advanced microvascular complications, hypoglycemia unawareness, and individual patient considerations.
Factors thatinterfere with HbA1c Measurement:Hemoglobin variants, elevated fetal hemoglobin (HbF) and chemically modified
derivatives of hemoglobin (e.g. carbamylated Hb in patients with renal failure) can affect the accuracy of HbA1c measurements.
Factors thataffect interpretation ofHbA1c Measurement:Any condition that shortens erythrocyte survival or decrease mean
erythrocyte age (e. g., recovery from acute blood loss, hemolytic anemia, HbSS, HbCC, and HbSC) will falsely lower HbA1c test
results regardless of the assay method used. Iron deficiency anemia is associated with higher HbA1c.
Note: Presence of Hemoglobin variants and/or conditions that affect red cell turnover must be considered, particularly when the
HbA1c result does not correlate with the patient's blood glucose levels.
Page 3 of 14
This test has been performed at
TATA 1MG BANGALORE
Address: No 607, Ground, 1st & 2nd Floor, 80
Feet Road, 6th Block, Koramangala,
Bengaluru, 560095
Page 4 of 14
PO No :PO3758436865-720
BIOCHEMISTRY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Lipid Profile
Cholesterol - Total 149 mg/dL Desirable <170 Enzymatic
Borderline high 170–199
High >=200
Triglycerides 103 mg/dL Normal: <75, Glycerol Phosphate
Borderline: 75 - 99, Oxidase
High:>=100
Cholesterol - HDL 30 mg/dL Low HDL: <40 mg/dL Accelerator Selective
Borderline Low: 40-45 Detergent
mg/dL
Acceptable: >45 mg/dL
Cholesterol - LDL 98 mg/dl Acceptable: <110 mg/dLCalculated
Borderline High: 110-129
mg/dL
High: > or =130 mg/dL
Cholesterol- VLDL 21 mg/dl <15 Calculated
Cholesterol : HDL Cholesterol 5.0 Ratio Desirable : 3.5-4.5 Calculated
High Risk : >5
LDL : HDL Cholesterol 3.28 Ratio Desirable : 2.5-3.0 Calculated
High risk : >3.5
Non HDL Cholesterol 119 mg/dL Acceptable: <120 mg/dLCalculated
Borderline High: 120-144
mg/dL
High: >=145 mg/dL
HDL : LDL Ratio 0.30 Ratio >0.3 Calculated
Comment:
• Lipid profile measurements in the same patient can show physiological & analytical variations. It is recommended that 3 serial
samples 1 week apart may be tested.
• Indians are at a high risk of developing atherosclerotic cardiovascular disease (ASCVD); at a much earlier age and more severe
with high mortality. Dyslipidemia (abnormal lipid profile) is the major risk factor and found in almost 80% Indians.
•Total cholesterolis the total amount of cholesterol in blood comprising of HDL, LDL-C, and VLDL.
•LDL Cholesterol (LDL-C) or “bad”cholesterol contributes most significantly to atherosclerosis leading to heart disease or
stroke and is the primary target for reducing risk for cardiovascular disease.
•High-density lipoprotein (HDL)or “good” cholesterol can lower risk of heart disease and stroke.
Page 5 of 14
This test has been performed at
TATA 1MG BANGALORE
Address: No 607, Ground, 1st & 2nd Floor, 80
Feet Road, 6th Block, Koramangala,
Bengaluru, 560095
Page 6 of 14
PO No :PO3758436865-720
BIOCHEMISTRY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Bilirubin-Total 0.40 mg/dL 0.3-1.2 Diazonium Salt
Bilirubin-Direct 0.14 mg/dL 0.05-0.20 Diazo
Bilirubin-Indirect 0.26 mg/dL 0.2-0.8 Calculated
Protein, Total 7.60 g/dL 6.0-8.0 Biuret
Albumin 4.40 g/dL 3.8-5.4 Bromocresol Green
Globulin 3.2 g/dL 2.1 - 3.2 Calculated
A/G Ratio 1.38 Ratio 1.27 - 1.99 Calculated
Aspartate Transaminase (SGOT) 24 U/L 18-36 NADH w/o P-5’-P
SGPT (Alanine Transaminase) 11 U/L 9-25 NADH w/o P-5’-P
SGOT/SGPT 2.18 Ratio Calculated
Alkaline Phosphatase 188 U/L <500 Para-Nitrophenyl
Phosphate
Gamma Glutamyltransferase (GGT) 12 U/L 12-55 L-gamma-glutamyl-3-
Carboxy-4-Nitroanilide
Comment:
Raised ALT and AST indicate hepatocellular damage (e.g. viral or drugs etc). ALT is more liver-specific while AST is also
found in heart, skeletal muscle, and kidney. Mild elevation (less than twice normal) often resolves on its own. Fatty liver
disease (especially with metabolic syndrome) is a common cause in asymptomatic cases. Certain drugs (paracetamol,
statins), herbal supplements, energy drinks, and antibiotics may also affect liver function.
SGOT/SGPT Ratio: Typically <1 in healthy individuals (vary between 0.7-1.4; higher in women than men). High SGPT (ratio
<1) seen in acute or chronic hepatitis, autoimmune disorders, medications, toxins while ratio >1 indicates alcoholic
hepatitis, cirrhosis, metastasis or non-hepatic issues (hemolytic diseases, CVS disorders).
Elevated Alkaline Phosphatase and GGT: Suggest cholestatic diseases (e.g. bile duct obstruction, primary biliary
cirrhosis etc.) and can also be due to bone disease, pregnancy, chronic renal failure, malignancy, and congestive heart
failure.
High Bilirubin: Indicates jaundice due to increased RBC breakdown, liver damage (e.g., infections, toxins), or cholestasis
(e.g., gallstones, tumors ).
High Protein Levels: Seen in dehydration (e.g., severe vomiting, diarrhea) or increased production (e.g., inflammation,
hematopoietic neoplasms). Low protein and albumin: Result from impaired synthesis (liver disease), decreased intake,
tissue damage, malabsorption, or increased renal excretion.
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PO No :PO3758436865-720
BIOCHEMISTRY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
*Please note test values may vary depending on the assay method used.
Comment:
BUN is directly related to protein intake and nitrogen metabolism and inversely related to the rate of excretion of urea.Blood
urea nitrogen (BUN) levels reflect the balance between the production and excretion of urea. Increased levels are seen in renal
failure (acute or chronic), urinary tract obstruction, dehydration, shock, burns, CHF, GI bleeding, nephrotoxic drugs. Decreased
levels are seen in hepatic failure, nephrotic syndrome, cachexia (low-protein and high-carbohydrate diets).
Urea is a non-proteinous nitrogen compound formed in the liver from ammonia as an end product of protein metabolism. Urea
diffuses freely into extracellular and intracellular fluid and is ultimately excreted by the kidneys. Increased levels are found in
acute renal failure, chronic glomerulonephritis, congestive heart failure, decreased renal perfusion, diabetes, excessive protein
ingestion, gastrointestinal (GI) bleeding, hyperalimentation, hypovolemia, ketoacidosis, muscle wasting from starvation,
neoplasms, pyelonephritis, shock, urinary tract obstruction, nephrotoxic drugs. Decreased levels are seen in inadequate dietary
protein, low-protein/high-carbohydrate diet, malabsorption syndromes, pregnancy, severe liver disease, certain drugs.
Creatinine is catabolic product of creatinine phosphate, which is excreted by filtration through the glomerulus and by tubular
secretion. Creatinine clearance is an acceptable clinical measure of glomerular filtration rate (GFR). Increased levels are seen in
acute/chronic renal failure, urinary tract obstruction, hypothyroidism, nephrotoxic drugs, shock, dehydration, congestive heart
failure, diabetes. Decreased levels are found in muscular dystrophy.
BUN/Creatinine ratio(normally 12:1–20:1) is decreased in acute tubular necrosis, advanced liver disease, low protein intake,
and following hemodialysis. BUN/Creatinine ratio is increased in dehydration, GI bleeding, and increased catabolism.
Uric acid levels show diurnal variation. The level is usually higher in the morning and lower in the evening. Increased levels are
seen in starvation, strenuous exercise, malnutrition, or lead poisoning, gout, renal disorders, increased breakdown of body cells
in some cancers (including leukemia, lymphoma, and multiple myeloma) or cancer treatments, hemolytic anemia, sickle cell
anemia, or heart failure, pre-eclampsia, liver disease (cirrhosis), obesity, psoriasis, hypothyroidism, low blood levels of
parathyroid hormone (PTH), certain drugs, foods that are very high in purines - such as organ meats, red meats, some seafood
and beer. Decreased levels are seen in liver disease, Wilson's disease, Syndrome of inappropriate antidiuretic hormone (SIADH),
certain drugs.
Page 8 of 14
PO No :PO3758436865-720
BIOCHEMISTRY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Comment:
Iron is an essential trace mineral element which forms an important component of hemoglobin, metallocompounds and Vitamin
A. Deficiency of iron is seen in iron deficiency and anaemia of chronic disorders.
Increased iron concentration are seen in hemolytic anaemias, hemochromatosis and acute liver disease. Serum Iron alone is
unreliable due to considerable physiologic diurnal variation in the results with highest values in the morning and lowest values in
the evening as well as variation in response to iron therapy .
Total Iron Binding capacity (TIBC)is a direct measure of the protein Transferrin which transports iron from the gut to storage
sites in the bone marrow. Increased levels of TIBC suggest that total iron body stores are low, increased concentration may be
the sign of Iron deficiency anaemia, polycythemia vera ,and may occur during the third trimester of pregnancy. Decreased levels
may be seen in hemolytic anaemia, hemochromatosis, chronic liver disease, hypoproteinemia ,malnutrition.
Unsaturated Iron Binding Capacity (UIBC) is increased in low iron state and decreased in high iron concentration such as
hemochromatosis. In case of anaemia of chronic disease the patient may be anaemic but has adequate iron reserve and a low
uIBC.
Transferrin Saturationoccurs in Idiopathic hemochromatosis and Transfusional hemosiderosis where no unsaturated iron
binding capacity is available for iron mobilization. Similar condition is seen in congenital deficiency of Transferrin.
Page 9 of 14
This test has been performed at
TATA 1MG BANGALORE
Address: No 607, Ground, 1st & 2nd Floor, 80
Feet Road, 6th Block, Koramangala,
Bengaluru, 560095
Page 10 of 14
This test has been performed at
TATA 1MG BANGALORE
Address: No 607, Ground, 1st & 2nd Floor, 80
Feet Road, 6th Block, Koramangala,
Bengaluru, 560095
Page 11 of 14
PO No :PO3758436865-720
IMMUNOLOGY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Vitamin D deficiency is a cause of secondary hyperparathyroidism and diseases resulting in impaired bone metabolism (like
rickets, osteomalacia).
Recently, many chronic diseases such as cancer, high blood pressure, osteoporosis and several autoimmune diseases
have been linked to vitamin D deficiency.
The assay measures both D2 (Ergocalciferol) and D3 (Cholecalciferol) metabolites of vitamin D
Vitamin B12
Vitamin B12 660.0 pg/mL 187-833 CMIA
Comment:
Vitamin B12 along with folate is essential for DNA synthesis and myelin formation.
Decreased levels are seen in anaemia, term pregnancy, vegetarian diet, intrinsic factor deficiency, partial
gastrectomy/ileal damage, celiac disease, oral contraceptive use, parasitic infestation, pancreatic deficiency, treated
epilepsy, smoking, hemodialysis and advanced age.
Increased levels are seen in renal failure, hepatocelluar disorders, myeloproliferative disorders and at times with excess
supplementation of vitamins pills.
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PO No :PO3758436865-720
CLINICAL PATHOLOGY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Comment:
•Note: Pre-test condition to be observed while submitting the sample-first void, mid stream urine, collected in a clean, dry, sterile
container is recommended for routine urine analysis, avoid contamination with any discharge from vaginal, urethra, perineum,
Avoid prolonged transit time & undue exposure to sunlight.
•During interpretation, points to be considered are Negative nitrite test does not exclude the urinary tract infections. Trace
proteinuria can be seen with many physiological conditions like prolonged recumbency, exercise, high protein diet. False positive
reactions for bile pigments, proteins, glucose and nitrites can be caused by peroxidase like activity by disinfectants, therapeutic
dyes, ascorbic acid and certain drugs. • Urine microscopy is done in centrifuged urine specimens
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PO No :PO3758436865-720
CLINICAL PATHOLOGY
SUPER EMPLOYEE HEALTH CHECK-UP
Test Name Result Unit Bio. Ref. Interval Method
Disclaimer : Results relate only to the sample received. Test results marked "BOLD" indicate abnormal results i.e. higher or lower than normal. All
lab test results are subject to clinical interpretation by a qualified medical professional. This report cannot be used for any medico-legal purposes.
Partial reproduction of the test results is not permitted. Also, TATA 1mg Labs is not responsible for any misinterpretation or misuse of the
information. The test reports alone may not be conclusive of the disease/condition, hence clinical correlation is necessary. Reports should be
vetted by a qualified doctor only.
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Ensuring accuracy IN every single report
Following a 3-step review process:
Verified test
External assessments Trained phlebotomists
procedures
Thorough third-party Ensuring smooth sample
Highly standardized test
assessment by authorized collection experience &
procedures following CLSI*
experts pre-analytical precision
guidelines
*Clinical & Laboratory Standards Institute T&C Apply